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The Biology of Retinopathy of Prematurity How Knowledge of Pathogenesis Guides Treatment

Journal article
Authors L. E. Smith
Anna-Lena Hård
Ann Hellström
Published in Clinics in Perinatology
Volume 40
Issue 2
Pages 201–214
ISSN 0095-5108
Publication year 2013
Published at Institute of Neuroscience and Physiology, Department of Clinical Neuroscience and Rehabilitation
Pages 201–214
Language en
Links dx.doi.org/10.1016/j.clp.2013.02.00...
Keywords Oxygen, IGF-1, Omega-3 polyunsaturated fatty acids, Postnatal weight gain, Erythropoietin, endothelial growth-factor, oxygen-induced retinopathy, postnatal, weight-gain, aggressive posterior retinopathy, screening algorithm, winrop, retinal neovascularization, preterm infants, proliferative, retinopathy, igf-i, bevacizumab avastin
Subject categories Ophthalmology, Pediatrics

Abstract

Retinopathy of prematurity occurs because the retina of a preterm infant at birth is incompletely vascularized, and if the postnatal environment does not match the in utero environment that supported retinal development, the vessels and neural retina will not grow normally. Risk factors determined from many clinical studies and animal studies fall into 2 categories: prenatal factors and postnatal factors.

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