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C. elegans ten-1 is synthetic lethal with mutations in cytoskeleton regulators, and enhances many axon guidance defective mutants.

Journal article
Authors Catarina Mörck
Vivekanand Vivekanand
Gholamali Jafari
Marc Pilon
Published in BMC developmental biology
Volume 10
Pages 55
ISSN 1471-213X
Publication year 2010
Published at Department of Cell and Molecular Biology
Pages 55
Language en
Links dx.doi.org/10.1186/1471-213X-10-55
Keywords Animals, Axons, metabolism, Caenorhabditis elegans, genetics, metabolism, Caenorhabditis elegans Proteins, genetics, metabolism, Membrane Proteins, genetics, metabolism, Mutation, Pharynx, innervation
Subject categories Cell and molecular biology, Molecular biology, Cell Biology, Morphology, Genetics, Developmental Biology, Neurobiology

Abstract

Teneurins are transmembrane proteins that assist morphogenetic processes in many organisms. ten-1 is the C. elegans teneurin homolog with two transcripts, ten-1a and ten-1b, that respectively encode a long (TEN-1L) and short (TEN-1S) form of the protein. We previously isolated a C. elegans mutant where one pharyngeal neuron was frequently misplaced, and now show that it corresponds to a novel allele of ten-1.

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