To the top

Page Manager: Webmaster
Last update: 9/11/2012 3:13 PM

Tell a friend about this page
Print version

C. elegans ten-1 is synth… - University of Gothenburg, Sweden Till startsida
To content Read more about how we use cookies on

C. elegans ten-1 is synthetic lethal with mutations in cytoskeleton regulators, and enhances many axon guidance defective mutants.

Journal article
Authors Catarina Mörck
Vivekanand Vivekanand
Gholamali Jafari
Marc Pilon
Published in BMC developmental biology
Volume 10
Pages 55
ISSN 1471-213X
Publication year 2010
Published at Department of Cell and Molecular Biology
Pages 55
Language en
Keywords Animals, Axons, metabolism, Caenorhabditis elegans, genetics, metabolism, Caenorhabditis elegans Proteins, genetics, metabolism, Membrane Proteins, genetics, metabolism, Mutation, Pharynx, innervation
Subject categories Cell and molecular biology, Molecular biology, Cell Biology, Morphology, Genetics, Developmental Biology, Neurobiology


Teneurins are transmembrane proteins that assist morphogenetic processes in many organisms. ten-1 is the C. elegans teneurin homolog with two transcripts, ten-1a and ten-1b, that respectively encode a long (TEN-1L) and short (TEN-1S) form of the protein. We previously isolated a C. elegans mutant where one pharyngeal neuron was frequently misplaced, and now show that it corresponds to a novel allele of ten-1.

Page Manager: Webmaster|Last update: 9/11/2012

The University of Gothenburg uses cookies to provide you with the best possible user experience. By continuing on this website, you approve of our use of cookies.  What are cookies?