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No support for premature central nervous system aging in HIV-1 when measured by cerebrospinal fluid phosphorylated tau (p-tau).

Journal article
Authors Jan Jessen Krut
Richard W Price
Henrik Zetterberg
Dietmar Fuchs
Lars Hagberg
Aylin Yilmaz
Paola Cinque
Staffan Nilsson
Magnus Gisslén
Published in Virulence
Volume 8
Issue 5
Pages 599-604
ISSN 2150-5608
Publication year 2017
Published at Institute of Biomedicine, Department of Medical and Clinical Genetics
Institute of Neuroscience and Physiology, Department of Psychiatry and Neurochemistry
Institute of Biomedicine, Department of Infectious Medicine
Pages 599-604
Language en
Links dx.doi.org/10.1080/21505594.2016.12...
Subject categories Neurochemistry


The prevalence of neurocognitive deficits are reported to be high in HIV-1 positive patients, even with suppressive antiretroviral treatment, and it has been suggested that HIV can cause accelerated aging of the brain. In this study we measured phosphorylated tau (p-tau) in cerebrospinal fluid (CSF) as a potential marker for premature central nervous system (CNS) aging. P-tau increases with normal aging but is not affected by HIV-associated neurocognitive disorders.

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