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Cardiac arrest in Wilson's disease after curative liver transplantation: a life-threatening complication of myocardial copper excess?

Journal article
Authors Niklas Forsgard
Anders Oldfors
Entela Bollano
Bert Andersson
Niklas Bergh
Kristjan Karason
Christian Lars Polte
Published in ESC Heart Failure
Volume 6
Issue 1
Pages 228-231
ISSN 2055-5822
Publication year 2019
Published at Institute of Biomedicine
Institute of Medicine
Pages 228-231
Language en
Links dx.doi.org/10.1002/ehf2.12395
Keywords Wilson disease, Copper, Cardiac arrest, Ventricular arrhythmia
Subject categories Cardiovascular medicine

Abstract

We report the case of a 38-year-old man who presented with cardiac arrest 1 year after curative liver transplantation for Wilson's disease. Clinical work-up proofed myocardial copper and iron accumulation using mass spectrometry, which led most likely to myocardial fibrosis as visualized by cardiovascular magnetic resonance (unprecedented delayed enhancement pattern) and endomyocardial biopsy. Consequently, cardiac arrest due to ventricular fibrillation and subsequent episodes of sustained ventricular tachycardia were considered as primary cardiac manifestation of Wilson's disease. This can, as illustrated by our case, occur even late after curative liver transplantation, which is an important fact that treating physicians should be aware of during clinical follow-up of these patients.

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Denna text är utskriven från följande webbsida:
http://www.gu.se/english/research/publication/?publicationId=278511
Utskriftsdatum: 2019-08-25